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ABCC1-mediated corticosterone export protects from glucocorticoid-induced cardiometabolic toxicity
Nixon, Mark
(Principal Investigator)
Stimson, Roland
(Co-investigator)
School of Neurological and Cardiovascular Sciences
Overview
Fingerprint
Research output
(1)
Project Details
Status
Finished
Effective start/end date
1/07/22
→
31/12/25
Funding
British Heart Foundation:
£199,270.21
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Fingerprint
Explore the research topics touched on by this project. These labels are generated based on the underlying awards/grants. Together they form a unique fingerprint.
Cortisol
Biochemistry, Genetics and Molecular Biology
100%
Deficiency
Biochemistry, Genetics and Molecular Biology
100%
Insulin Resistance
Biochemistry, Genetics and Molecular Biology
100%
Corticosterone
Biochemistry, Genetics and Molecular Biology
100%
Glucocorticoid
Biochemistry, Genetics and Molecular Biology
88%
Energy Expenditure
Biochemistry, Genetics and Molecular Biology
11%
Dose Response
Biochemistry, Genetics and Molecular Biology
11%
Wild Type Mouse
Biochemistry, Genetics and Molecular Biology
11%
Research output
Research output per year
2026
2026
2026
1
Article
Research output per year
Research output per year
Abcc1 deficiency protects from corticosterone but not cortisol-induced adiposity and insulin resistance in a sex-specific manner
Paul, M. A., Nagy, B., Laforest, S.,
Simpson, J. P.
, Mungall, W.,
Homer, N. Z. M.
,
Semple, R. K.
,
Andrew, R.
,
Stimson, R. H.
&
Nixon, M.
,
4 Mar 2026
, (E-pub ahead of print)
In:
The Journal of Physiology.
Research output
:
Contribution to journal
›
Article
›
peer-review
Open Access
File
Insulin Resistance
100%
Cortisol
100%
Corticosterone
100%
Deficiency
100%
Glucocorticoid
88%