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Cause and Consequences of Mitochondrial Injury in Progressive Multiple Sclerosis
Mahad, Don
(Principal Investigator)
Deanery of Clinical Sciences
Centre for Clinical Brain Sciences
Anne Rowling Regenerative Neurology Clinic
Edinburgh Neuroscience
Overview
Fingerprint
Research output
(1)
Project Details
Status
Finished
Effective start/end date
1/08/14
→
31/12/15
Funding
Non-EU-based charities:
£57,591.00
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Fingerprint
Explore the research topics touched on by this project. These labels are generated based on the underlying awards/grants. Together they form a unique fingerprint.
Pyramidal Cell
Biochemistry, Genetics and Molecular Biology
100%
Farnesyltransferase
Biochemistry, Genetics and Molecular Biology
100%
Granule Cell
Biochemistry, Genetics and Molecular Biology
100%
Synapse
Biochemistry, Genetics and Molecular Biology
100%
Heme
Neuroscience
100%
Mitochondrial Function
Neuroscience
100%
Electron Transport Chain
Biochemistry, Genetics and Molecular Biology
60%
Cytochrome C Oxidase
Biochemistry, Genetics and Molecular Biology
60%
Research output
Research output per year
2017
2017
2017
1
Article
Research output per year
Research output per year
Loss of protohaem IX farnesyltransferase in mature dentate granule cells impairs short-term facilitation at mossy fibre to CA3 pyramidal cell synapses
Booker, S. A.
, Campbell, G. R., Mysiak, K. S.,
Brophy, P. J.
,
Kind, P. C.
,
Mahad, D. J.
&
Wyllie, D. J. A.
,
15 Mar 2017
,
In:
The Journal of Physiology.
595
,
6
,
p. 2147-2160
14 p.
Research output
:
Contribution to journal
›
Article
›
peer-review
Open Access
File
Farnesyltransferase
100%
Synapse
100%
Granule Cell
100%
Pyramidal Cell
100%
Mitochondrial Function
100%