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Characterisation of FAM38A, a novel integrin activator in lung cancer
McHugh, Brian
(Principal Investigator)
Sethi, Tariq
(Co-investigator)
Deanery of Clinical Sciences
Centre for Inflammation Research
Overview
Fingerprint
Research output
(1)
Project Details
Status
Finished
Effective start/end date
1/04/09
→
30/04/11
Funding
UK central government bodies/local authorities, health and hospital authorities:
£256,743.00
View all
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Fingerprint
Explore the research topics touched on by this project. These labels are generated based on the underlying awards/grants. Together they form a unique fingerprint.
Cell Migration
Biochemistry, Genetics and Molecular Biology
100%
Membrane Protein
Biochemistry, Genetics and Molecular Biology
100%
Integrin
Biochemistry, Genetics and Molecular Biology
100%
Transmembrane Protein
Biochemistry, Genetics and Molecular Biology
100%
Transmembrane Domain
Biochemistry, Genetics and Molecular Biology
11%
Cell Adhesion
Biochemistry, Genetics and Molecular Biology
11%
Overall Survival
Biochemistry, Genetics and Molecular Biology
11%
Survival Rate
Biochemistry, Genetics and Molecular Biology
11%
Research output
Research output per year
2012
2012
2012
1
Article
Research output per year
Research output per year
Loss of the integrin-activating transmembrane protein Fam38A (Piezo1) promotes a switch to a reduced integrin-dependent mode of cell migration
McHugh, B. J., Murdoch, A.,
Haslett, C.
& Sethi, T.,
Jul 2012
,
In:
PLoS ONE.
7
,
7
,
7 p.
, e40346.
Research output
:
Contribution to journal
›
Article
›
peer-review
Open Access
File
Cell Migration
100%
Membrane Protein
100%
Transmembrane Protein
100%
Integrin
100%
Cell Adhesion
11%