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Determination of T cell signalling pathway alterations induced by the neutrophil peptide LL-37
Gwyer Findlay, Emily
(Principal Investigator)
Deanery of Clinical Sciences
University of Edinburgh
Overview
Fingerprint
Research output
(2)
Project Details
Status
Finished
Effective start/end date
1/10/17
→
30/09/18
Funding
UK-based charities:
£11,800.00
View all
View less
Fingerprint
Explore the research topics touched on by this project. These labels are generated based on the underlying awards/grants. Together they form a unique fingerprint.
T Cell
Biochemistry, Genetics and Molecular Biology
100%
Cathelicidin
Biochemistry, Genetics and Molecular Biology
88%
Antimicrobial Peptides
Biochemistry, Genetics and Molecular Biology
88%
Cell Contact
Biochemistry, Genetics and Molecular Biology
88%
Polypeptide Antibiotic Agent
Medicine and Dentistry
88%
Neutrophil
Medicine and Dentistry
88%
Cell Proliferation
Biochemistry, Genetics and Molecular Biology
17%
Programmed Cell Death
Medicine and Dentistry
12%
Research output
Research output per year
2021
2021
2021
2
Article
Research output per year
Research output per year
The Outcome of Neutrophil-T Cell Contact Differs Depending on Activation Status of Both Cell Types
Minns, D.
,
Smith, K. J.
,
Hardisty, G.
,
Rossi, A. G.
&
Gwyer Findlay, E.
,
30 Mar 2021
,
In:
Frontiers in Immunology.
12
Research output
:
Contribution to journal
›
Article
›
peer-review
Open Access
File
Neutrophil
100%
T Cell
100%
Cell Contact
100%
T Cell Proliferation
20%
Cell Proliferation
20%
The neutrophil antimicrobial peptide cathelicidin promotes Th17 differentiation
Minns, D.
,
Smith, K. J.
,
Alessandrini, V.
,
Hardisty, G.
,
Melrose, L.
,
Jackson-Jones, L.
,
MacDonald, A. S.
,
Davidson, D. J.
&
Gwyer Findlay, E. L.
,
24 Feb 2021
,
In:
Nature Communications.
Research output
:
Contribution to journal
›
Article
›
peer-review
Open Access
File
Polypeptide Antibiotic Agent
100%
Neutrophil
100%
Cathelicidin
100%
Antimicrobial Peptides
100%
T Cell
14%