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Investigating the binding partners of the C9orf72 protein through immunoprecipitation and mass spectrometry
Mehta, Arpan
(Principal Investigator)
University of Edinburgh
Overview
Fingerprint
Research output
(1)
Project Details
Status
Finished
Effective start/end date
1/05/19
→
1/02/21
Funding
UK central government bodies/local authorities, health and hospital authorities:
£15,000.00
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Fingerprint
Explore the research topics touched on by this project. These labels are generated based on the underlying awards/grants. Together they form a unique fingerprint.
Motor Neuron
Neuroscience
100%
Amyotrophic Lateral Sclerosis
Neuroscience
100%
Phenotype
Neuroscience
28%
Mitochondrial Function
Neuroscience
28%
Axon
Neuroscience
28%
Gene Expression
Neuroscience
14%
Sensory Neuron
Neuroscience
14%
Mitochondrial Biogenesis
Neuroscience
14%
Research output
Research output per year
2021
2021
2021
1
Article
Research output per year
Research output per year
Mitochondrial bioenergetic deficits in C9orf72 amyotrophic lateral sclerosis motor neurons cause dysfunctional axonal homeostasis
Mehta, A. R.
,
Gregory, J. M.
,
Dando, O.
,
Carter, R. N.
,
Burr, K.
,
Nanda, J.
,
Story, D.
,
Mcdade, K.
,
Smith, C.
,
Morton, N. M.
,
Mahad, D. J.
,
Hardingham, G. E.
,
Chandran, S.
&
Selvaraj, B. T.
,
4 Jan 2021
,
In:
Acta Neuropathologica.
Research output
:
Contribution to journal
›
Article
›
peer-review
Open Access
File
Motor Neuron
100%
Amyotrophic Lateral Sclerosis
100%
Axon
28%
Mitochondrial Function
28%
Phenotype
28%