Can we manipulate the ovary’s own metabolism to protect it from chemotherapy-induced damage?

Adomas Liugaila, Agnes Stefansdottir, Norah Spears*

*Corresponding author for this work

Research output: Contribution to journalBook/Film/Article reviewpeer-review

Abstract

Some chemotherapy treatments induce female infertility through accelerated ovarian ageing, including due to nicotinamide adenine dinucleotide (NAD) depletion. Using various mouse models, Ho et al (2024) demonstrate that exposure to two such chemotherapy drugs, cisplatin or doxorubicin, deplete ovarian NAD, with levels restored by administrating the exogenous NAD precursor nicotinamide mononucleotide, ameliorating the drugs’ damaging effects on fertility.
Original languageEnglish
Pages (from-to)2274-2275
JournalEMBO Molecular Medicine
Volume16
Issue number10
DOIs
Publication statusPublished - 3 Sept 2024

Fingerprint

Dive into the research topics of 'Can we manipulate the ovary’s own metabolism to protect it from chemotherapy-induced damage?'. Together they form a unique fingerprint.

Cite this