Molecular mechanisms of ovarian hyperstimulation syndrome: paracrine reduction of endothelial claudin 5 by hCG in vitro is associated with increased endothelial permeability

M Rodewald; D Herr; W C Duncan; H M Fraser; G Hack; R Konrad; F Gagsteiger; R Kreienberg; C Wulff

doi:10.1093/humrep/den479

Molecular mechanisms of ovarian hyperstimulation syndrome: paracrine reduction of endothelial claudin 5 by hCG in vitro is associated with increased endothelial permeability

M Rodewald, D Herr, W C Duncan, H M Fraser, G Hack, R Konrad, F Gagsteiger, R Kreienberg, C Wulff

Research output: Contribution to journal › Article › peer-review

Abstract

Ovarian hyperstimulation syndrome (OHSS) is a potentially life-threatening complication of ovarian stimulation associated with severe vascular hyperpermeability. Primary co-cultures of human luteinized granulosa cells (LGCs) and human umbilical vein endothelial cells (HUVECs) were used as a model of steroidgenic/endothelial cell interaction in OHSS.

Original language	English
Pages (from-to)	1191-9
Number of pages	9
Journal	Human Reproduction
Volume	24
Issue number	5
DOIs	https://doi.org/10.1093/humrep/den479
Publication status	Published - May 2009

Access to Document

10.1093/humrep/den479

Cite this

Rodewald, M., Herr, D., Duncan, W. C., Fraser, H. M., Hack, G., Konrad, R., Gagsteiger, F., Kreienberg, R., & Wulff, C. (2009). Molecular mechanisms of ovarian hyperstimulation syndrome: paracrine reduction of endothelial claudin 5 by hCG in vitro is associated with increased endothelial permeability. Human Reproduction, 24(5), 1191-9. https://doi.org/10.1093/humrep/den479

@article{a67feb2d1d904f82b24e56d7204a7870,

title = "Molecular mechanisms of ovarian hyperstimulation syndrome: paracrine reduction of endothelial claudin 5 by hCG in vitro is associated with increased endothelial permeability",

abstract = "Ovarian hyperstimulation syndrome (OHSS) is a potentially life-threatening complication of ovarian stimulation associated with severe vascular hyperpermeability. Primary co-cultures of human luteinized granulosa cells (LGCs) and human umbilical vein endothelial cells (HUVECs) were used as a model of steroidgenic/endothelial cell interaction in OHSS.",

author = "M Rodewald and D Herr and Duncan, {W C} and Fraser, {H M} and G Hack and R Konrad and F Gagsteiger and R Kreienberg and C Wulff",

year = "2009",

month = may,

doi = "10.1093/humrep/den479",

language = "English",

volume = "24",

pages = "1191--9",

journal = "Human Reproduction",

issn = "0268-1161",

publisher = "Oxford University Press",

number = "5",

}

Rodewald, M, Herr, D, Duncan, WC, Fraser, HM, Hack, G, Konrad, R, Gagsteiger, F, Kreienberg, R & Wulff, C 2009, 'Molecular mechanisms of ovarian hyperstimulation syndrome: paracrine reduction of endothelial claudin 5 by hCG in vitro is associated with increased endothelial permeability', Human Reproduction, vol. 24, no. 5, pp. 1191-9. https://doi.org/10.1093/humrep/den479

Molecular mechanisms of ovarian hyperstimulation syndrome : paracrine reduction of endothelial claudin 5 by hCG in vitro is associated with increased endothelial permeability. / Rodewald, M; Herr, D; Duncan, W C; Fraser, H M; Hack, G; Konrad, R; Gagsteiger, F; Kreienberg, R; Wulff, C.

In: Human Reproduction, Vol. 24, No. 5, 05.2009, p. 1191-9.

Research output: Contribution to journal › Article › peer-review

TY - JOUR

T1 - Molecular mechanisms of ovarian hyperstimulation syndrome

T2 - paracrine reduction of endothelial claudin 5 by hCG in vitro is associated with increased endothelial permeability

AU - Rodewald, M

AU - Herr, D

AU - Duncan, W C

AU - Fraser, H M

AU - Hack, G

AU - Konrad, R

AU - Gagsteiger, F

AU - Kreienberg, R

AU - Wulff, C

PY - 2009/5

Y1 - 2009/5

N2 - Ovarian hyperstimulation syndrome (OHSS) is a potentially life-threatening complication of ovarian stimulation associated with severe vascular hyperpermeability. Primary co-cultures of human luteinized granulosa cells (LGCs) and human umbilical vein endothelial cells (HUVECs) were used as a model of steroidgenic/endothelial cell interaction in OHSS.

AB - Ovarian hyperstimulation syndrome (OHSS) is a potentially life-threatening complication of ovarian stimulation associated with severe vascular hyperpermeability. Primary co-cultures of human luteinized granulosa cells (LGCs) and human umbilical vein endothelial cells (HUVECs) were used as a model of steroidgenic/endothelial cell interaction in OHSS.

UR - http://www.scopus.com/inward/record.url?scp=62549111347&partnerID=8YFLogxK

U2 - 10.1093/humrep/den479

DO - 10.1093/humrep/den479

M3 - Article

C2 - 19168871

VL - 24

SP - 1191

EP - 1199

JO - Human Reproduction

JF - Human Reproduction

SN - 0268-1161

IS - 5

ER -

Molecular mechanisms of ovarian hyperstimulation syndrome: paracrine reduction of endothelial claudin 5 by hCG in vitro is associated with increased endothelial permeability

Abstract

Access to Document

Other files and links

Fingerprint

Cite this