Reactivation and mutation of newly discovered WU, KI, and Merkel cell carcinoma polyomaviruses in immunosuppressed individuals

Colin P Sharp, Päivi Norja, Iain Anthony, Jeanne E Bell, Peter Simmonds

Research output: Contribution to journalArticlepeer-review

Abstract

Infection with the human polyomaviruses BK (BKV) and JC (JCV) is almost ubiquitous, asymptomatic, and lifelong. However, reactivation during immunosuppression, associated with mutations in the transcriptional control region (TCR) that up-regulates viral replication, can cause life-threatening disease. In this study, we investigated whether the recently discovered WU and KI polyomaviruses (WUPyV and KIPyV) and Merkel cell polyomavirus (MCPyV) could, like BKV and JCV, persist, mutate, and reactivate in immunodeficient subjects.
Original languageEnglish
Pages (from-to)398-404
Number of pages7
JournalThe Journal of Infectious Diseases
Volume199
Issue number3
DOIs
Publication statusPublished - Feb 2009

Fingerprint

Dive into the research topics of 'Reactivation and mutation of newly discovered WU, KI, and Merkel cell carcinoma polyomaviruses in immunosuppressed individuals'. Together they form a unique fingerprint.

Cite this