Regulatory T cells: a brake on ischemic injury or an active promoter of tissue healing?

David A. Ferenbach; David C. Kluth; Jeremy Hughes

doi:10.1038/ki.2009.302

Regulatory T cells: a brake on ischemic injury or an active promoter of tissue healing?

Research output: Contribution to journal › Editorial › peer-review

Abstract

Gandolfo et al. report the key role played by regulatory T cells in modulating recovery from ischemic renal injury. The role of regulatory T cells in modulating ischemia-reperfusion injury in the kidney and other organs is discussed in this commentary.

Original language	English
Pages (from-to)	689-691
Number of pages	4
Journal	Kidney International
Volume	76
Issue number	7
DOIs	https://doi.org/10.1038/ki.2009.302
Publication status	Published - Oct 2009

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10.1038/ki.2009.302

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title = "Regulatory T cells: a brake on ischemic injury or an active promoter of tissue healing?",

abstract = "Gandolfo et al. report the key role played by regulatory T cells in modulating recovery from ischemic renal injury. The role of regulatory T cells in modulating ischemia-reperfusion injury in the kidney and other organs is discussed in this commentary.",

author = "Ferenbach, \{David A.\} and Kluth, \{David C.\} and Jeremy Hughes",

year = "2009",

month = oct,

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journal = "Kidney International",

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AU - Hughes, Jeremy

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N2 - Gandolfo et al. report the key role played by regulatory T cells in modulating recovery from ischemic renal injury. The role of regulatory T cells in modulating ischemia-reperfusion injury in the kidney and other organs is discussed in this commentary.

AB - Gandolfo et al. report the key role played by regulatory T cells in modulating recovery from ischemic renal injury. The role of regulatory T cells in modulating ischemia-reperfusion injury in the kidney and other organs is discussed in this commentary.

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DO - 10.1038/ki.2009.302

M3 - Editorial

C2 - 19752862

SN - 0085-2538

VL - 76

SP - 689

EP - 691

JO - Kidney International

JF - Kidney International

IS - 7

ER -

Regulatory T cells: a brake on ischemic injury or an active promoter of tissue healing?

Abstract

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