Using genetic variation to disentangle the complex relationship between food intake and health outcomes

Nicola Pirastu, Ciara McDonnell, Eryk Jan Grzeszkowiak, Ninon Mounier, Fumiaki Imamura, Jordi Merino, Felix R Day, Jie Zheng, Nele Taba, Maria Pina Concas, Linda Repetto, Katherine Kentistou, Antonietta Robino, Tõnu Esko, Peter K Joshi, Krista Fischer, Ken K. Ong, Tom R Gaunt, Zoltan Kutalik, John R. B. PerryJames F Wilson

Research output: Contribution to journalArticlepeer-review

Abstract

Diet is considered as one of the most important modifiable factors influencing human health, but efforts to identify foods or dietary patterns associated with health outcomes often suffer from biases, confounding, and reverse causation. Applying Mendelian randomization in this context may provide evidence to strengthen causality in nutrition research. To this end, we first identified 283 genetic markers associated with dietary intake in 445,779 UK Biobank participants. We then converted these associations into direct genetic effects on food exposures by adjusting them for effects mediated via other traits. The SNPs which did not show evidence of mediation were then used for MR, assessing the association between genetically predicted food choices and other risk factors, health outcomes. We show that using all associated SNPs without omitting those which show evidence of mediation, leads to biases in downstream analyses (genetic correlations, causal inference), similar to those present in observational studies. However, MR analyses using SNPs which have only a direct effect on the exposure on food exposures provided unequivocal evidence of causal associations between specific eating patterns and obesity, blood lipid status, and several other risk factors and health outcomes.

Original languageEnglish
Article numbere1010162
JournalPLoS Genetics
Volume18
Issue number6
Early online date2 Jun 2022
DOIs
Publication statusPublished - Jun 2022

Keywords / Materials (for Non-textual outputs)

  • Causality
  • Eating
  • Genetic Variation
  • Humans
  • Outcome Assessment, Health Care
  • Risk Factors

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