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A targeted mutation disrupting mitochondrial complex IV function in primary afferent neurons leads to pain hypersensitivity through P2Y1 receptor activation.

Research output: Contribution to journalArticle

Original languageEnglish
JournalMolecular Neurobiology
Early online date28 Jan 2019
DOIs
Publication statusE-pub ahead of print - 28 Jan 2019

ID: 78775635