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Activation of hypoxia-inducible factor-1 alpha (Hif-1 alpha) delays inflammation resolution by reducing neutrophil apoptosis and reverse migration in a zebrafish inflammation model

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Activation of hypoxia-inducible factor-1 alpha (Hif-1 alpha) delays inflammation resolution by reducing neutrophil apoptosis and reverse migration in a zebrafish inflammation model. / Elks, Philip M.; van Eeden, Fredericus J.; Dixon, Giles; Wang, Xingang; Reyes-Aldasoro, Constantino Carlos; Ingham, Philip W.; Whyte, Moira K. B.; Walmsley, Sarah R.; Renshaw, Stephen A.

In: Blood, Vol. 118, No. 3, 21.07.2011, p. 712-722.

Research output: Contribution to journalArticle

Harvard

Elks, PM, van Eeden, FJ, Dixon, G, Wang, X, Reyes-Aldasoro, CC, Ingham, PW, Whyte, MKB, Walmsley, SR & Renshaw, SA 2011, 'Activation of hypoxia-inducible factor-1 alpha (Hif-1 alpha) delays inflammation resolution by reducing neutrophil apoptosis and reverse migration in a zebrafish inflammation model', Blood, vol. 118, no. 3, pp. 712-722. https://doi.org/10.1182/blood-2010-12-324186

APA

Elks, P. M., van Eeden, F. J., Dixon, G., Wang, X., Reyes-Aldasoro, C. C., Ingham, P. W., ... Renshaw, S. A. (2011). Activation of hypoxia-inducible factor-1 alpha (Hif-1 alpha) delays inflammation resolution by reducing neutrophil apoptosis and reverse migration in a zebrafish inflammation model. Blood, 118(3), 712-722. https://doi.org/10.1182/blood-2010-12-324186

Vancouver

Elks PM, van Eeden FJ, Dixon G, Wang X, Reyes-Aldasoro CC, Ingham PW et al. Activation of hypoxia-inducible factor-1 alpha (Hif-1 alpha) delays inflammation resolution by reducing neutrophil apoptosis and reverse migration in a zebrafish inflammation model. Blood. 2011 Jul 21;118(3):712-722. https://doi.org/10.1182/blood-2010-12-324186

Author

Elks, Philip M. ; van Eeden, Fredericus J. ; Dixon, Giles ; Wang, Xingang ; Reyes-Aldasoro, Constantino Carlos ; Ingham, Philip W. ; Whyte, Moira K. B. ; Walmsley, Sarah R. ; Renshaw, Stephen A. / Activation of hypoxia-inducible factor-1 alpha (Hif-1 alpha) delays inflammation resolution by reducing neutrophil apoptosis and reverse migration in a zebrafish inflammation model. In: Blood. 2011 ; Vol. 118, No. 3. pp. 712-722.