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Insufficient OPC migration into demyelinated lesions is a cause of poor remyelination in MS and mouse models

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Insufficient OPC migration into demyelinated lesions is a cause of poor remyelination in MS and mouse models. / Boyd, Amanda; Zhang, Hongyan; Williams, Anna.

In: Acta Neuropathologica, 06.2013, p. 1-19.

Research output: Contribution to journalArticle

Harvard

Boyd, A, Zhang, H & Williams, A 2013, 'Insufficient OPC migration into demyelinated lesions is a cause of poor remyelination in MS and mouse models', Acta Neuropathologica, pp. 1-19. https://doi.org/10.1007/s00401-013-1112-y

APA

Boyd, A., Zhang, H., & Williams, A. (2013). Insufficient OPC migration into demyelinated lesions is a cause of poor remyelination in MS and mouse models. Acta Neuropathologica, 1-19. https://doi.org/10.1007/s00401-013-1112-y

Vancouver

Boyd A, Zhang H, Williams A. Insufficient OPC migration into demyelinated lesions is a cause of poor remyelination in MS and mouse models. Acta Neuropathologica. 2013 Jun;1-19. https://doi.org/10.1007/s00401-013-1112-y

Author

Boyd, Amanda ; Zhang, Hongyan ; Williams, Anna. / Insufficient OPC migration into demyelinated lesions is a cause of poor remyelination in MS and mouse models. In: Acta Neuropathologica. 2013 ; pp. 1-19.