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Precision engineering for PRRSV resistance in pigs: Macrophages from genome edited pigs lacking CD163 SRCR5 domain are fully resistant to both PRRSV genotypes while maintaining biological function

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Precision engineering for PRRSV resistance in pigs: Macrophages from genome edited pigs lacking CD163 SRCR5 domain are fully resistant to both PRRSV genotypes while maintaining biological function. / Burkard, Christine; Lillico, Simon; Reid, Elizabeth; Jackson, Ben; Mileham, Alan J.; Ait-Ali, Tahar; Whitelaw, Christopher; Archibald, Alan.

In: Plos pathogens, Vol. 13, No. 2, e1006206, 23.02.2017.

Research output: Contribution to journalArticle

Harvard

Burkard, C, Lillico, S, Reid, E, Jackson, B, Mileham, AJ, Ait-Ali, T, Whitelaw, C & Archibald, A 2017, 'Precision engineering for PRRSV resistance in pigs: Macrophages from genome edited pigs lacking CD163 SRCR5 domain are fully resistant to both PRRSV genotypes while maintaining biological function', Plos pathogens, vol. 13, no. 2, e1006206. https://doi.org/10.1371/journal.ppat.1006206

APA

Burkard, C., Lillico, S., Reid, E., Jackson, B., Mileham, A. J., Ait-Ali, T., ... Archibald, A. (2017). Precision engineering for PRRSV resistance in pigs: Macrophages from genome edited pigs lacking CD163 SRCR5 domain are fully resistant to both PRRSV genotypes while maintaining biological function. Plos pathogens, 13(2), [e1006206]. https://doi.org/10.1371/journal.ppat.1006206

Vancouver

Burkard C, Lillico S, Reid E, Jackson B, Mileham AJ, Ait-Ali T et al. Precision engineering for PRRSV resistance in pigs: Macrophages from genome edited pigs lacking CD163 SRCR5 domain are fully resistant to both PRRSV genotypes while maintaining biological function. Plos pathogens. 2017 Feb 23;13(2). e1006206. https://doi.org/10.1371/journal.ppat.1006206

Author

Burkard, Christine ; Lillico, Simon ; Reid, Elizabeth ; Jackson, Ben ; Mileham, Alan J. ; Ait-Ali, Tahar ; Whitelaw, Christopher ; Archibald, Alan. / Precision engineering for PRRSV resistance in pigs: Macrophages from genome edited pigs lacking CD163 SRCR5 domain are fully resistant to both PRRSV genotypes while maintaining biological function. In: Plos pathogens. 2017 ; Vol. 13, No. 2.